Prescription medicine reference

Tesamorelin

trans-3-hexenoyl-GHRH(1-44) amide · a stabilised growth hormone-releasing hormone analogue

A 44-amino-acid synthetic analogue of human GHRH, modified at the N-terminus to resist enzymatic degradation.

FDA approved (2010) — HIV-associated lipodystrophy

Residues

44

Formula

C221H366N72O67S

Molecular weight

~5135.9 g/mol

Class

GHRH analogue

YADAIFTNSYRKVLGQLSARKLLQDIMSRQQGESNQERGARARL

Clinical evidence

Unlike research peptides, Tesamorelin has phase 3 randomised trial data — but only for one indication.

Visceral adipose tissue reduction

The approved indication — significant VAT reduction versus placebo in phase 3 trials in people with HIV-associated lipodystrophy.

Phase 3 RCT

IGF-1 elevation

Consistent, dose-related increase.

Phase 3 RCT

Triglyceride reduction

Improvement observed in trial populations.

Secondary endpoint

Regain on discontinuation

Visceral fat returns after treatment stops, indicating continuous administration is required for maintained effect.

Phase 3 RCT

Glucose tolerance

A recognised concern; GH elevation can worsen insulin sensitivity, and glucose parameters require monitoring.

Phase 3 RCT

NAFLD / liver fat

Investigated for hepatic fat reduction in HIV populations with promising results, but not an approved indication.

Phase 2Investigational

Cognition and other uses

Explored in small investigational studies; not approved and not established.

Investigational

Sequence

44 residues, N-terminus to C-terminus, wrapped 11 per row on desktop with continuous numbering. Both termini are modified.

N-term · trans-3-hexenoyl cap

Blocks DPP-4 cleavage — the entire point of the molecule.

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C-term · amide (-NH₂)

Hover or focus a residue to inspect. Use class filters to isolate hydrophobic, basic or acidic residues along the chain.

Sequence: YADAIFTNSYRKVLGQLSARKLLQDIMSRQQGESNQERGARARL. 44 residues, N-terminally acylated with a trans-3-hexenoyl group and C-terminally amidated.

Amino acid composition

How the 44 positions distribute across 16 unique residues.

44residues

By class

Basic
8
Acidic
4
Aromatic
3
Hydrophobic
14
Polar
12
Structural
3

16 of 20 proteinogenic amino acids are present. Absent: Cys, His, Pro, Trp.

Arg13.6%

Arginine

× 6pos 11, 20, 29, 38, 41, 43
Ala11.4%

Alanine

× 5pos 2, 4, 19, 40, 42
Gln11.4%

Glutamine

× 5pos 16, 24, 30, 31, 36
Leu11.4%

Leucine

× 5pos 14, 17, 22, 23, 44
Ser9.1%

Serine

× 4pos 9, 18, 28, 34
Gly6.8%

Glycine

× 3pos 15, 32, 39
Asn4.5%

Asparagine

× 2pos 8, 35
Asp4.5%

Aspartic acid

× 2pos 3, 25
Glu4.5%

Glutamic acid

× 2pos 33, 37
Ile4.5%

Isoleucine

× 2pos 5, 26
Lys4.5%

Lysine

× 2pos 12, 21
Tyr4.5%

Tyrosine

× 2pos 1, 10
Met2.3%

Methionine

× 1pos 27
Phe2.3%

Phenylalanine

× 1pos 6
Thr2.3%

Threonine

× 1pos 7
Val2.3%

Valine

× 1pos 13

Structural properties

Five readouts characterising the chain and its modifications.

Readout · 01

N-terminal hexenoyl modification

The defining feature. A trans-3-hexenoyl group blocks DPP-4 cleavage and substantially extends half-life relative to native GHRH.

Readout · 02

Net charge ≈ +4

Eight basic residues (6× Arg, 2× Lys) against four acidic. Both termini are blocked: acylated at the N-terminus, amidated at the C-terminus.

Readout · 03

No cysteine

No disulphide bridges; structure is stabilised by helical propensity rather than cross-links.

Readout · 04

No proline

Nothing interrupting the backbone, consistent with the amphipathic α-helix the central region is thought to adopt on receptor binding.

Readout · 05

Single methionine at position 27

An oxidation-sensitive residue and a known formulation stability consideration.

Mechanism of action

Well-characterised — this is a labelled prescription drug.

Established mechanism
Tesamorelin
GHRH receptor on pituitary somatotrophs
Pulsatile endogenous GH release
Hepatic IGF-1 production
Lipolysis · preferentially visceral adipose
Tesamorelin stimulates the body's own pulsatile GH secretion rather than supplying exogenous growth hormone — a pharmacologically meaningful distinction, since the pituitary's own feedback control remains partly intact.

Regulatory status

Approved medicine.

Approved by the FDA in 2010 under the brand name Egrifta, with later reformulations (Egrifta SV, Egrifta WR). A genuine, licensed prescription drug.

Narrow indication.

Approved specifically for the reduction of excess abdominal fat in adults with HIV-associated lipodystrophy. Not approved for general fat loss, bodybuilding, anti-ageing or athletic use.

Prescription only.

Requires prescription and medical supervision, including monitoring of IGF-1 and glucose. Material sold outside pharmacy channels is not the licensed product.

Prohibited in sport.

As a growth hormone secretagogue it is prohibited by WADA at all times under category S2 (peptide hormones, growth factors and related substances).

Contraindicated in pregnancy, in active malignancy, and in disruption of the hypothalamic-pituitary axis — this is a drug with a real risk profile, not a wellness compound.